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Department of Cardiovascular Sciences
University of Leicester
Clinical Sciences Wing
Glenfield General Hospital
Leicester
LE3 9QP

General Enquiries to
Louise Goddard
Tel: 0116 258 3045

Postgraduate General Enquiries to
Postgraduate Tutor

For other contact details see under
Research Groups
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Dr Glenn Rodrigo

Lecturer in cardiovascular sciences

B.Sc. (Biological Sciences), University of Leicester, 1980
Ph.D. (Physiology), University of Leicester, 1984

Tel: 0116 258 3028

Email: gcr4@le.ac.uk

Address: Department of Cardiovascular Sciences, University of Leicester, Clinical Sciences Wing, Glenfield General Hospital, Leicester, LE3 9QP

For general enquiries please email the secretary, Louise Goddard, or telephone 0116 256 3405

Research Interests

  • Intracellular ion regulation in cardiac muscle in health and disease.
  • Remote ischaemic pre- and post-conditioning of cardiac muscle during acute and chronic heart disease: Translational studies in humans.
  • Circadian rhythms and the cardiovascular system: Influence of neuronal nitric oxide synthase on excitation-contraction coupling in ventricular cardiomyocyte and vascular smooth muscle.

Membership of Learned Societies

The Physiological Society of the UK 1987-
The European Working Group on Cardiac Cellular Electrophysiology 1988-

Key Publications

Collins HE, Rodrigo GC. Inotropic Response of Cardiac Ventricular Myocytes to beta-Adrenergic Stimulation With Isoproterenol Exhibits Diurnal Variation. Involvement of Nitric Oxide. Circ Res. 2010 Apr 16 106 7 1244-52

Rodrigo GC, Samani NJ. Ischemic preconditioning of the whole heart confers protection on subsequently isolated ventricular myocytes. Am J Physiol Heart Circ Physiol. 2008 Jan 294 1 H524-31

Hardy ME, Lawrence CL, Standen NB, Rodrigo GC. Can optical recordings of membrane potential be used to screen for drug-induced action potential prolongation in single cardiac myocytes? J Pharmacol Toxicol Methods. 2006 Sep-Oct 54 2 173-82

Rodrigo GC, Standen NB. Role of mitochondrial re-energization and Ca2+ influx in reperfusion injury of metabolically inhibited cardiac myocytes. Cardiovasc Res. 2005 Aug 1 67 2 291-300

Rodrigo GC, Standen NB. ATP-sensitive potassium channels. Curr Pharm Des. 2005  11 15 1915-40

Rodrigo GC, Davies NW, Standen NB. Diazoxide causes early activation of cardiac sarcolemmal KATP channels during metabolic inhibition by an indirect mechanism. Cardiovasc Res. 2004 Feb 15 61 3 570-9

Rodrigo GC, Lawrence CL, Standen NB. Dinitrophenol pretreatment of rat ventricular myocytes protects against damage by metabolic inhibition and reperfusion. J Mol Cell Cardiol. 2002 May 34 5 555-69

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